Source: American Journal of Physiology Heart and Circulatory Physiology. Unidade: FCFRP
Subjects: ÓXIDO NÍTRICO, ENDOTÉLIO, GLUTAMATOS
ABNT
CUNHA, Natalia Veronez da et al. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats. American Journal of Physiology Heart and Circulatory Physiology, v. 306, n. 10, p. H1472-H1480, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00520.2013. Acesso em: 04 maio 2024.APA
Cunha, N. V. da, Pinge-Filho, P., Panis, C., Silva, B. R., Pernomian, L., Grando, M. D., et al. (2014). Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats. American Journal of Physiology Heart and Circulatory Physiology, 306( 10), H1472-H1480. doi:10.1152/ajpheart.00520.2013NLM
Cunha NV da, Pinge-Filho P, Panis C, Silva BR, Pernomian L, Grando MD, Cecchini R, Bendhack LM, Martins-Pinge MC. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats [Internet]. American Journal of Physiology Heart and Circulatory Physiology. 2014 ; 306( 10): H1472-H1480.[citado 2024 maio 04 ] Available from: https://doi.org/10.1152/ajpheart.00520.2013Vancouver
Cunha NV da, Pinge-Filho P, Panis C, Silva BR, Pernomian L, Grando MD, Cecchini R, Bendhack LM, Martins-Pinge MC. Decreased endothelial nitric oxide, systemic oxidative stress, and increased sympathetic modulation contribute to hypertension in obese rats [Internet]. American Journal of Physiology Heart and Circulatory Physiology. 2014 ; 306( 10): H1472-H1480.[citado 2024 maio 04 ] Available from: https://doi.org/10.1152/ajpheart.00520.2013